Bystander activation and autoimmunity

The interaction over time of genetic, epigenetic and environmental factors (i.e., autoimmune ecology) increases or decreases the liability an individual would have to develop an autoimmune disease (AD) depending on the misbalance between risk and protective effects. Pathogens have been the most comm...

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Autores:

Tipo de recurso:

Fecha de publicación:

2019

Institución:

Universidad del Rosario

Repositorio:

Repositorio EdocUR - U. Rosario

Idioma:

eng

OAI Identifier:

oai:repository.urosario.edu.co:10336/22244

Acceso en línea:

https://doi.org/10.1016/j.jaut.2019.06.012
https://repository.urosario.edu.co/handle/10336/22244

Palabra clave:

Autoimmune disease
Autoimmune hepatitis
Autoimmune thyroiditis
Autoimmunity
Bacterial infection
Bacterium
Bystander effect
Cell communication
Gap junction
Graves disease
Hashimoto disease
Host
Human
Immunological tolerance
Insulin dependent diabetes mellitus
Latent period
Legionella pneumophila
Mediator
Memory t lymphocyte
Multiple sclerosis
Nonhuman
Parasitosis
Priority journal
Review
Rheumatoid arthritis
Systemic lupus erythematosus
T lymphocyte activation
Vaccination
Virus infection
Auto-reactive t cells
Autoimmune diseases
Bystander activation
Cytokines
Infection
T-cell activation

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