Infections and vaccines in the etiology of antiphospholipid syndrome

Purpose of review: To present scientific evidence supporting the infectious origin for the antiphospholipid syndrome (APS) by molecular mimicry between pathogens, infection and vaccination with ?2-glycoprotein I (?2-GPI) molecule. Recent findings: APS is characterized by the presence of pathogenic a...

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Autores:

Tipo de recurso:

Fecha de publicación:

2012

Institución:

Universidad del Rosario

Repositorio:

Repositorio EdocUR - U. Rosario

Idioma:

eng

OAI Identifier:

oai:repository.urosario.edu.co:10336/23823

Acceso en línea:

https://doi.org/10.1097/BOR.0b013e32835448b8
https://repository.urosario.edu.co/handle/10336/23823

Palabra clave:

4 aminobutyric acid
Aluminum hydroxide
Beta2 glycoprotein 1
Beta2 glycoprotein 1 antibody
Cardiolipin antibody
Diphtheria antibody
Ganglioside antibody
Glycerol
Immunoglobulin a antibody
Immunoglobulin g antibody
Immunoglobulin m antibody
Interleukin 1 receptor
Laminin
Myeloid differentiation factor 88
Tetanus toxoid
Toll like receptor
Antibody production
Antiphospholipid syndrome
Autoinflammatory disease
Blood clotting time
Clostridium tetani
Cross reaction
Enzyme linked immunosorbent assay
Fetus wastage
Human
Infection
Medical history
Molecular mimicry
Nonhuman
Partial thromboplastin time
Priority journal
Review
Signal transduction
Tetanus prophylaxis
Thrombocytopenia
Antiphospholipid syndrome
Autoantibodies
Beta 2-glycoprotein i
Cross reactions
Humans
Infection
Molecular mimicry
Tetanus toxoid
?2-glycoprotein i
Antiphospholipid syndrome
Tetanus toxoid
immunologic
Adjuvants

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