HIV replication is associated to inflammasomes activation, IL-1ß, IL-18 and caspase-1 expression in GALT and peripheral blood

Background Human immunodeficiency virus (HIV) promotes an inflammatory process, leading to the progressive loss of the functional capacity of the immune system. The HIV infection induces alterations in several tissues, but mainly in the gut-associated lymphoid tissue (GALT). However, the degree of G...

Full description

Autores:

Feria M.G.
Taborda N.A.
Hernández López, Juan Carlos
Rugeles M.T.

Tipo de recurso:

Article of journal

Fecha de publicación:

2018

Institución:

Universidad Cooperativa de Colombia

Repositorio:

Repositorio UCC

Idioma:

OAI Identifier:

oai:repository.ucc.edu.co:20.500.12494/41917

Acceso en línea:

https://doi.org/10.17843/rpmesp.2018.351.3568.
https://hdl.handle.net/20.500.12494/41917

Palabra clave:

apoptosis regulatory protein
calcium binding protein
caspase recruitment domain signaling protein
inflammasome
interleukin 18
interleukin 18 protein
human
interleukin 1beta
interleukin 1beta converting enzyme
NLRC4 protein
human
NLRP1 protein
human
PYCARD protein
human
signal transducing adaptor protein
adult
blood
CD4+ T lymphocyte
genetics
host pathogen interaction
human
Human immunodeficiency virus infection
lymphoid tissue
metabolism
mononuclear cell
physiology
rectum
virology
virus load
virus replication
Adaptor Proteins
Signal Transducing
Adult
Apoptosis Regulatory Proteins
Calcium-Binding Proteins
CARD Signaling Adaptor Proteins
Caspase 1
CD4-Positive T-Lymphocytes
HIV Infections
Host-Pathogen Interactions
Humans
Inflammasomes
Interleukin-18
Interleukin-1beta
Leukocytes
Mononuclear
Lymphoid Tissue
Rectum
Viral Load
Virus Replication

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License

http://purl.org/coar/access_right/c_14cb